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The evidence [1,7,9-11] included the infrarenal occlusion of the inferior caval vein, left colic artery and vein occlusion ischemic/reperfusion ulcerative colitis, superior anterior pancreaticoduodenal vein-induced duodenal venous congestion lesions, bile duct ligation-induced liver cirrhosis and portal hypertension, temporary occlusion of the portal triad (Pringle maneuver)-induced ischemia-reperfusion injury[10], and suprahepatic occlusion of the inferior caval vein-induced Budd-Chiari syndrome[11]
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ROS may be important mediators of cellular injury during COVID-19 sepsis (Silvagno et al., 2020), either following macromolecular damage or by hindering extracellular and intracellular regulatory processes
