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In a study focused on astrocytes, researchers found that NF‐KB singling is more likely to be activated in the Nrf2 knockout cells than the wild‐type cells do and lead to more pro‐inflammatory factors released in an mechanical scratch model.93 Furthermore, a variety of other inflammatory signalling pathways have been found to be inhibited by activating Nrf2 signal pathway in astrocytes.54, 141 5.2