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Similarly, inhibition of ROS by butylated hydroxyanisole suppressed activation of TNF-induced necroptosis, suggesting the critical role of ROS in this form of cellular death (Lin et al., 2004)
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Studies on the metabolic consequences of K-Ras activation in a mouse model of pancreatic cancer revealed that the nonoxidative PPP is substantially activated, whereas the oxidative branch is unaffected 29
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