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The peptide mimics the FOXO4 binding domain, displacing endogenous FOXO4 and releasing p53 to restore apoptotic signaling selectively in senescent cells where FOXO4 overexpression creates the competitive binding environment
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PHR1 and PHR2 are pH-responsive genes regulated by Rim101, with PHR2 being required for virulence at acidic infection sites (such as the vaginal tract), whereas PHR1 is required at alkaline sites of infection (such as the bloodstream) [167]
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