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What sustained research interest beyond its initial discovery was an unexpected characteristic: DSIP didn’t simply sedate — it appeared to modulate the quality of sleep architecture, particularly slow-wave delta activity, while simultaneously influencing stress hormones and pain signaling

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For chloramphenicol, BAA/245 was susceptible and PAO1 was intermediate-resistant, while for colistin, BAA/245 was resistant and PAO1 was susceptible