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These activated microglia stay in this phenotype and damage neurons by causing the release of pro-inflammatory cytokines such as tumor necrosis factor-α (TNF-α), and interleukin-1β (IL-1β) [146,149,152,153,154,155,156,157,158,159]
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Kim OH, Kang GH, Noh H, Cha JY, Lee HJ, Yoon JH, et al
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