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Importantly, finding a measurable serum concentration of acetaminophen 24 hours or more after the last dose should suggest the possibility of acetaminophen hepatotoxicity

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The underlying genetic basis for this reduction involves mutations or polymorphisms in the UGT family 1 member A1 ( UGT1A1 ) gene, with homozygosity for the (TA)7TAA variant in the promoter region being particularly prevalent in white populations[15]

is known for its ability to inhibit tyrosinase