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Although the exact mechanism is unknown, high GSH levels are likely protecting cells against reactive oxygen species and are facilitating the conjugation and elimination of reactive intermediates or signaling molecules (Vlachaki and Meyn, 1998)

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In the presence of inflammatory cytokines such as TNF-α, treatment with CQ elevated caspase-3 and cleaved poly (adenosine diphosphate-ribose) polymerases, indicating enhanced osteoblast apoptosis (50,77)